Diabrix

Rs. 999.00 Rs. 699.00 (30% OFF)

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1) How Diabrix lowers blood sugar & HbA1c — the biology (mechanisms)

A. Increases insulin sensitivity (peripheral tissue)

• Several botanical compounds activate AMP-activated protein kinase (AMPK) or related pathways in liver and muscle → increased glucose uptake and fatty-acid oxidation (the same metabolic node targeted by metformin). Berberine is a well-studied example (mechanistic and human data).

B. Protects and/or stimulates pancreatic β-cells

• Gymnema and some other botanicals show insulinotropic effects (increase insulin release) and can protect β-cells from cytokine-induced damage in preclinical and early human studies. This helps lower postprandial glucose and longer-term glycosylation (HbA1c).

C. Slows carbohydrate absorption (post-meal glucose blunting)

• Fibrous or enzyme-inhibiting herb components (fenugreek, bitter melon fractions, cinnamon polyphenols) reduce α-glucosidase activity or form viscous complexes in the gut → lower postprandial glucose spikes (important for HbA1c). Fenugreek has consistent trial support for this.

D. Anti-inflammatory & antioxidant effects (preserve insulin signalling)

• Chronic inflammation and oxidative stress worsen insulin resistance. Curcumin, Guduchi, and other herbs reduce inflammatory cytokines and oxidative damage, improving insulin signalling and β-cell health—effects which translate to improved glycemic indexes over weeks–months.

E. Gut microbiome & metabolic hormones

• Botanical mixtures (Triphala/fenugreek/bitter melon etc.) change gut microbiota and increase short-chain fatty acids (SCFAs) which help insulin sensitivity and incretin hormones — a downstream route to lower fasting glucose and HbA1c. Clinical formulations that modulate microbiome have improved metabolic markers in trials.

2) What clinical studies show (real patient data → HbA1c improvements)

Below are representative human evidence points for common ingredients/formulations (these are examples of the “type” of evidence supporting Diabrix-style blends):

• Berberine — meta-analyses and RCTs show berberine lowers fasting glucose, improves HbA1c and lipids; effect sizes are clinically meaningful and safety profile is acceptable as adjunct therapy. (systematic reviews/meta-analysis).

• Fenugreek (Trigonella) — randomized and controlled trials show reductions in fasting glucose and HbA1c; fenugreek also lowers triglycerides and body measures in some studies.

• Cinnamon — meta-analyses of RCTs show small but significant reductions in fasting glucose and a modest improvement in HbA1c (~0.09% average across trials included in older meta-analysis); effects depend on dose and study length.

• Curcumin — randomized trials and systematic reviews show curcumin or nano-curcumin can reduce FBG and HbA1c, and can improve β-cell function on longer supplementation.

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